Muaj ntau txoj hauv kev los kuaj ntshav qab zib. Txhua txoj kev feem ntau yuav tsum tau rov ua dua rau hnub thib ob txhawm rau kuaj ntshav qab zib.

Cov tsos mob ntawm ntshav qab zib muaj xws li polydipsia, polyuria, polyeating, thiab tsis piav qhia qhov hnyav.

Kev yoo mov ntshav qabzib, ntshav qabzib random, lossis OGTT 2h ntshav qabzib yog lub hauv paus tseem ceeb rau kev kuaj mob ntshav qab zib. Yog tias tsis muaj cov tsos mob tshwm sim ntawm cov ntshav qab zib mellitus, qhov kev sim yuav tsum tau rov ua dua kom paub meej qhov kev kuaj mob. (A) Hauv chav kuaj nrog kev tswj hwm nruj, HbA1C txiav txim siab los ntawm cov txheej txheem kev sim tuaj yeem siv los ua tus qauv kuaj mob ntxiv rau ntshav qab zib. (B) Raws li etiology, ntshav qab zib tau muab faib ua 4 hom: T1DM, T2DM, hom ntshav qab zib tshwj xeeb thiab ntshav qab zib gestational. (A)

Kev kuaj HbA1c ntsuas koj cov ntshav qabzib nruab nrab rau ob mus rau peb lub hlis dhau los. Qhov zoo ntawm kev kuaj mob no yog tias koj tsis tas yuav yoo mov lossis haus ib yam dab tsi.

Mob ntshav qab zib tau kuaj pom ntawm HbA1c ntau dua lossis sib npaug li 6.5%.

Peb Baysen kev kho mob tuaj yeem muab cov khoom siv kuaj HbA1c sai rau Kev kuaj mob ntshav qab zib thaum ntxov.Zoo siab txais tos hu rau cov lus qhia ntxiv.