Muaj ntau txoj kev los kuaj mob ntshav qab zib. Feem ntau txhua txoj kev yuav tsum tau ua dua rau hnub thib ob kom kuaj mob ntshav qab zib.

Cov tsos mob ntawm tus kab mob ntshav qab zib muaj xws li polydipsia, polyuria, polyeating, thiab poob phaus tsis paub meej.

Kev ntsuas ntshav qab zib thaum yoo mov, ntshav qab zib tsis raws cai, lossis OGTT 2 teev ntshav qab zib yog lub hauv paus tseem ceeb rau kev kuaj mob ntshav qab zib. Yog tias tsis muaj cov tsos mob ntawm ntshav qab zib, qhov kev kuaj mob yuav tsum tau rov ua dua kom paub meej tias tau kuaj mob. (A) Hauv chav kuaj mob uas muaj kev tswj hwm zoo, HbA1C txiav txim siab los ntawm cov txheej txheem kuaj mob txheem tuaj yeem siv ua tus qauv kuaj mob ntxiv rau ntshav qab zib. (B) Raws li etiology, ntshav qab zib tau muab faib ua 4 hom: T1DM, T2DM, ntshav qab zib hom tshwj xeeb thiab ntshav qab zib thaum cev xeeb tub. (A)

Qhov kev kuaj HbA1c ntsuas koj cov ntshav qab zib nruab nrab rau ob mus rau peb lub hlis dhau los. Qhov zoo ntawm kev kuaj mob li no yog tias koj tsis tas yuav yoo mov lossis haus dej dab tsi.

Kab mob ntshav qab zib raug kuaj pom thaum HbA1c ntau dua lossis sib npaug li 6.5%.

Peb Baysen kho mob tuaj yeem muab cov khoom siv kuaj mob HbA1c sai rau kev kuaj mob ntshav qab zib thaum ntxov. Txais tos tiv tauj kom paub meej ntxiv.